Extract
We read with great interest the work reported by Yoon et al. [1]. The authors found that a high concentration of nitrogen dioxide at an individual's residential address is significantly associated with increased mortality in idiopathic pulmonary fibrosis (IPF) patients when all confounders are included in the health analysis model. However, an important question that remains unclear in the study is whether active or passive cigarette smoking-associated release of nitrogen dioxide or particulate matter plays a role in the clinical outcome of IPF patients.
Abstract
The role of pollutants derived from cigarette smoke in the mortality of patients with pulmonary fibrosis https://bit.ly/3pbuPDc
Footnotes
Author contributions: Approval and writing the draft of the correspondence: T. Yasuma, C.N. D'Alessandro-Gabazza, O. Hataji, T. Kobayashi and E.C. Gabazza.
Conflict of interest: T. Yasuma reports grants from Shionogi, outside the submitted work.
Conflict of interest: C.N. D'Alessandro-Gabazza reports grants from Shionogi Pharmaceutical Inc., Astellas Pharmaceuticals Inc. and Asahi Kassei, outside the submitted work.
Conflict of interest: O. Hataji has nothing to disclose.
Conflict of interest: T. Kobayashi reports grants and personal fees from Chugai, Pfizer, ONO, Boehringer Ingelheim and Eli Lilly, grants from TAIHO, outside the submitted work.
Conflict of interest: E.C. Gabazza reports grants from Shionogi Pharmaceutical Inc., Astellas Pharmaceuticals Inc. and Asahi Kassei, outside the submitted work.
- Received May 15, 2021.
- Accepted May 28, 2021.
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